Index to Chiropractic Literature
Index to Chiropractic Literature
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Tuesday, April 23, 2024
Index to Chiropractic LiteratureIndex to Chiropractic LiteratureIndex to Chiropractic Literature
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ID 19287
  Title The organisation of the stress response, and its relevance to chiropractors: a commentary [editorial]
URL http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1629015/
Journal Chiropr & Osteopat. 2006 ;14(1):Online access only 54 p.
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Subject(s)
Peer Review No
Publication Type Article
Abstract/Notes The stress response is a natural reaction by the body, against potentially harmful stimuli to enhance the chance for survival. Persistent activation of the stress response can cause changes to homeostatic mechanisms. The study of stress neurophysiology, in the evaluation of the manifestation of disease in the body, suggests that these chronic changes have detrimental effects on sub cortical structures. Furthermore, there is much scientific support for the notion that chronic activation of supraspinal systems will lead to maladaptation of homeostatic mechanisms, causing the impairment of processes within the body, and ultimately leading to visceral disorders. The chiropractic profession for many years has alluded to chronic change of neurophysiological pathways as a potential explanation of visceral disorders, but the profession has typically described these in terms of somatovisceral or viscerosomatic reflex activity. Change in supraspinal neurophysiological efferent activity is increasingly being used to explain stress related disease. The chiropractic profession should consider investigating such stress responses by conducting spinal manipulative therapy trials that evaluate supraspinal effects of manipulation. Such research may help elucidate key mechanisms associated with the change of visceral disorders noted by some chiropractors following manipulative therapy. Index Terms: stress, HPA axis, psychoneuroimmunology, neurophysiology, disease, chiropractic, manipulation.

This abstract is reproduced with the permission of the publisher. Click on the above link for free full text. PubMed Record

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